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尼古丁檢測試劑盒 尼古丁檢測試劑卡
廣州健侖生物科技?有限公司
本司長期供應尼古丁(可替寧)檢測試劑盒,其主要品牌包括美國NovaBios、廣州健侖、廣州創侖等進口產品,國產產品,試劑盒的實驗方法是膠體金方法。
我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。
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【檢驗原理】本品采用競爭抑制法和膠體金免疫層析法原理定性檢測尿液中的尼古丁,以金標尼古丁單克隆抗體作為指示標記物,在硝酸纖維素膜上的檢測線處和控制線處分別包被尼古丁-BSA結合物和羊抗鼠IgG多克隆抗體。檢測時,尿樣在毛細效應下層析。如尿樣中的尼古丁濃度低于200ng/mL時,金標抗體不能全部與尼古丁結合,未結合的金標抗體在層析過程中與固定在檢測線處的尼古丁-BSA結合物結合,從而在檢測區(T)出現一條紫紅色條帶;如尿樣中尼古丁濃度高于200ng/mL時,金標抗體全部與尼古丁結合,從而在檢測區(T)因為競爭反應不會與尼古丁-BSA結合物結合而不出現紫紅色條帶。無論尿樣中是否存在尼古丁,控制區(C)都會出現一條紫紅色條帶。控制區(C)所呈現的紫紅色條帶是判斷是否有足夠的尿樣,層析過程是否正常的標準,同時也作為試劑的內控標準。
【主要組成成份】
檢測需要但未提供的材料:
【儲存條件及有效期】
尼古丁檢測試劑盒
儲存條件:原包裝應儲存于4~30℃避光干燥處,切忌冷凍。
有效期:24個月。
試劑盒應在鋁箔袋拆封后1小時內盡快使用;建議在周圍溫度高于30℃或高濕度條件下,盡可能做到即開即用。
【樣本要求】
【檢驗方法】
在進行檢測前必須先完整閱讀使用說明書,使用前將本品和尿樣恢復至室溫(20℃~30℃)。
尼古丁檢測試劑
急性型:臨床zui常見,患病豬腹瀉,糞便呈紅褐色,有的有組織碎片,病程一般維持48 h,36 h后死亡。
亞急性型:病程稍慢,癥狀特點為長時間腹瀉,病初排黃色糞便,之后逐漸變為含有組織碎片的稀便,病程維持5-7 d,zui后死亡。
慢性型:病程比較長,病例為時好時壞的間歇腹瀉,排灰黃色糞便,日漸消瘦,zui后常因生長停滯而淘汰,部分死亡。
臨床剖檢時zui常見癥狀為腸道病變,空腸病理變化zui為嚴重,腸道內容物液化,空腸內絨毛脫落壞死,腸系膜及漿膜下有大小不等的小氣泡,腸系膜淋巴結腫大,多數呈鮮紅色。病程長的動物以壞死性腸炎為主要臨床癥狀,腸粘膜易剝,腸腔內含大量壞死組織碎片。
C型產氣莢膜梭菌不僅感染豬,還可感染羊、雞、馬、兔和牛等動物。健康動物腸道內存在該菌,一旦動物腸道內的菌群失調,產氣莢膜梭菌大量繁殖和生長,排出的糞便污染了動物飼料或沒有。大量的該病菌就會隨著幼畜吸吮奶或食用污染物等方式進入幼畜體內,侵襲腸絨毛上皮并產生大量毒素,使腸道正常組織破損,毒素趁機而入,進而隨血液循環侵害其它組織器官。
實驗室中由C型產氣莢膜梭菌所導致的動物和人類的腸毒血癥和腸炎,與自然感染的動物臨床癥狀相似,病程也可分為亞急性、慢性和急性。其中急性和亞急性病例常表現為精神抑郁、劇烈腹痛和帶血稀便。慢性病例主要發生在成年動物中,其臨床表現為脫水和持續性腹瀉。
D型產氣莢膜梭菌能產生α、ε毒素,可導致成年牛的腸毒血癥、山羊的小腸和結腸炎、犢牛和羔羊的腸毒血癥(軟你好)等。腸毒血癥是由D型產氣莢膜梭菌而引起的一種重要的人畜共患病。該病的流行特點是發病急驟,死亡快,多呈散發,可以引起發病動物的大量死亡,嚴重制約了畜牧業的長遠發展。
感染D型產氣莢膜梭菌的綿羊和山羊兩者臨床表現不同,山羊主要表現為小腸結腸炎,而綿羊病理變化表現為心包積水、大腦和肺部水腫、腸道癥狀。以上兩者的實質器官病理變化相似。
?尼古丁檢測試劑
想了解更多的韓國SD產品及服務請掃描下方二維碼:我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。
二維碼掃一掃
【公司名稱】 廣州健侖生物科技有限公司
【】 楊永漢
【】
【騰訊 】
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-3室
【企業文化宣傳】
Acute type: the most common clinical, sick pigs diarrhea, brown stool, and some tissue fragments, the duration of the general maintenance of 48 h, died after 36 h.
Subacute type: a lesser duration, the symptoms characterized by prolonged diarrhea, the disease early row of yellow droppings, and then gradually become loose stools containing tissue fragments, duration of 5-7 d, the final death.
Chronic type: a longer course of illness, the case of intermittent diarrhea, good and bad times, discharge of yellow feces, getting thinner, and finally often due to stagnant growth and out, part of the death.
Clinical examination of the most common symptoms of intestinal disease, pathological changes in the most serious jejunum, intestinal contents of liquefaction, jejunum villi off necrosis, mesenteric and subserosal size of small bubbles, mesenteric lymph nodes, most were Red. Long duration of the disease to necrotizing enterocolitis as the main clinical symptoms, intestinal mucosa easy to peel, the intestine contains a large number of necrotic tissue fragments.
Clostridium perfringens type C not only infect pigs, but also in sheep, chickens, horses, rabbits and cattle and other animals. The bacteria exist in the intestine of healthy animals, and Clostridium perfringens multiplies and grows once the flora in the intestinal tract of the animal is out of control. The excreted faeces contaminate the animal feed or not. A large number of the bacteria will be with young animals suck milk or food contaminants into the young animals such as the body, invasion of intestinal villi and produce large amounts of toxins, the normal tissue damage to the intestine, toxin took the opportunity to enter, and then infringement of blood circulation with other Tissue organs.
Animal and human enterotoxemia and enteritis caused by C-type Clostridium perfringens in laboratory are similar to the clinical symptoms of naturally infected animals. The course of the disease can be divided into subacute, chronic and acute. The acute and subacute cases often show depression, severe abdominal pain and loose stools. Chronic cases occur mainly in adult animals, with clinical manifestations of dehydration and persistent diarrhea.
Clostridium perfringens type D can produce alpha, epsilon toxins that can cause enterotoxemia in adult cattle, small intestine and colitis in goats, enterotoxemia in calves and lambs (soft hello), and the like. Enterotoxemia is an important zoonosis caused by Clostridium perfringens type D. The prevalence of the disease is characterized by sudden onset, rapid death, mostly distributed, can cause a large number of deaths of animals, seriously hampered the long-term development of animal husbandry.
The clinical manifestations of both sheep and goat infected with Clostridium perfringens type D were different. Goat mainly manifested as enterocolitis. Pathological changes in sheep showed pericardial hydrops, brain and pulmonary edema, and intestinal symptoms. Both of the above two organs of the same pathological changes.?
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