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西班牙Certest艱難梭菌GDH重組蛋白
廣州健侖生物科技有限公司
廣州健侖長期供應各種生物原料，主要代理品牌：西班牙Certest。
主要產品包括各種生物單克隆抗原抗體、重組蛋白。
西班牙Certest艱難梭菌GDH重組蛋白
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【產品介紹】
| 貨號 | 產品名稱 | 規格 | 英文名稱 |
| MT-18EH30 | 阿米巴原蟲抗體（克隆H30） | x1mg | Anti-Entamoeba Mab (clone EH30) |
| MT-25ETV | 腸道病毒VP1重組蛋白 | x1mg | Enterovirus VP1 recombinant protein |
| MT-18EV5 | 腸道病毒抗體（克隆EV5） | x1mg | Anti-Enterovirus Mab (clone EV5) |
| MT-25STX | 大腸桿菌O157 VT1重組蛋白 | x1mg | E. coli O157 VT1 recombinant protein |
| MT-25VT2 | 大腸桿菌O157 VT2重組蛋白 | x1mg | E. coli O157 VT2 recombinant protein |
| MT-18E10 | 大腸桿菌O157抗體（克隆E10） | x1mg | Anti-E. coli O157 Mab (clone E10) |
| MT-18SN3 | 肺炎鏈球菌單克隆抗體(克隆SN3) | x1mg | Anti-Streptococcus pneumoniae Mab (clone SN3) |
| MT-18SN4 | 肺炎鏈球菌單克隆抗體(克隆SN4) | x1mg | Anti-Streptococcus pneumoniae Mab (clone SN4) |
| MT-16CP14 | 鈣結合蛋白單克隆抗體（克隆CP14） | x1mg | Anti-Calprotectin Mab (clone CP14) |
| MT-18RV3 | 呼吸道合胞病毒單抗（克隆RV3） | x1mg | Anti-RSV Mab (clone RV3) |
| MT-18RV4 | 呼吸道合胞病毒單抗（克隆RV4） | x1mg | Anti-RSV Mab (clone RV4) |
| MT-25RSV | 呼吸道合胞病毒重組融合蛋白 | x1mg | RSV recombinant fusion protein |
| MT-18Y77 | 甲型流感病毒單抗（克隆Y77） | x1mg | Anti-Influenza A Mab (clone Y77) |
| MT-25FAN | 甲型流感病毒重組核蛋白 | x1mg | Influenza A recombinant nucleoprotein |
| MT-16G18 | 賈第鞭毛蟲抗體（克隆G18） | x1mg | Anti-Giardia Mab trophozoite protein (clone G18) |
| MT-16G22 | 賈第鞭毛蟲抗體（克隆G22） | x1mg | Anti-Giardia Mab trophozoite protein (clone G22) |
| MT-25A1G | 賈第蟲腸道滋養體重組蛋白 | x1mg | Giardia intestinalis trophozoite recombinant protein |
| MT-25GCP | 賈第蟲腸囊菌重組蛋白 | x1mg | Giardia intestinalis cyst recombinant protein |
| MT-25GDH | 艱難梭菌GDH重組蛋白 | x1mg | Clostridium difficile GDH recombinant protein |
| MT-18TA5 | 艱難梭菌毒素A抗（克隆TA5） | x1mg | Anti-CD Toxin A Mab (clone TA5) |
| MT-18TA7 | 艱難梭菌毒素A抗（克隆TA7） | x1mg | Anti-CD Toxin A Mab (clone TA7) |
| MT-24TXA | 艱難梭菌毒素A重組蛋白（無毒性片段） | x1mg | C. difficile Toxin A recombinant protein (fragment without toxic activity) |
| MT-18TB41 | 艱難梭菌毒素B抗（克隆TB41） | x1mg | Anti-CD Toxin B Mab (clone TB41) |
| MT-18TB48 | 艱難梭菌毒素B抗（克隆TB48） | x1mg | Anti-CD Toxin B Mab (clone TB48) |
| MT-24TXB | 艱難梭菌毒素B重組蛋白（無毒性片段） | x1mg | C. difficile Toxin B recombinant protein (fragment without toxic activity) |
| MT-16GD10 | 艱難梭菌抗體（克隆GD10） | x1mg | Anti-GDH Mab (clone GD10) |
| MT-25CEP | 空腸彎曲桿菌重組外膜蛋白 | x1mg | Campylobacter jejuni recombinant outer membrane protein |
| MT-26VP6 | 輪狀病毒VP6重組蛋白 | x1mg | Rotavirus VP6 recombinant protein |
| MT-16R15 | 輪狀病毒單克隆抗體（克隆R15） | x1mg | Anti-Rotavirus Mab (clone R15) |
| MT-28SAGU | 滅活A鏈球菌抗原（天然提取物） | x1mg | Inactivated STREP A antigen (native extract) |
| MT-28SEU | 滅活腸炎沙門氏菌抗原（天然提取物） | x1mg | Inactivated Salmonella enteritidis antigen (native extract) |
| MT-28SBU | 滅活的鮑氏志賀氏菌抗原（天然提取物） | x1mg | Inactivated Shigella boydii antigen (native extract) |
| MT-28EC7U | 滅活的大腸桿菌O157抗原（天然提取物） | x1mg | Inactivated E. coli O157 antigen (native extract) |
| MT-28CCU | 滅活的大腸桿菌抗原（天然提取物） | x1mg | Inactivated Campylobacter coli antigen (native extract) |
| MT-28LMU | 滅活的單核細胞增生李斯特菌抗原（天然提取物） | x1mg | Inactivated Listeria monocytogenes antigen (native extract) |
| MT-28SPNU | 滅活的肺炎鏈球菌抗原（天然提取物） | x1mg | Inactivated Streptococcus pneumoniae antigen (native extract) |
| MT-28SFU | 滅活的福氏志賀氏菌抗原（天然提取物） | x1mg | Inactivated Shigella flexneri antigen (native extract) |
| MT-28CJU | 滅活的空腸彎曲桿菌抗原（天然提取物） | x1mg | Inactivated Campylobacter jejuni antigen (native extract) |
| MT-28SDU | 滅活的痢疾志賀氏菌抗原（天然提取物） | x1mg | Inactivated Shigella dysenteriae antigen (native extract) |
| MT-28LNU | 滅活的嗜肺軍團菌抗原（天然提取物） | x1mg | Inactivated Legionella pneumophila antigen (native extract) |
| MT-28STMU | 滅活的鼠傷寒沙門氏菌抗原（天然提取物） | x1mg | Inactivated Salmonella typhimurium antigen (native extract) |
| MT-28SSU | 滅活的宋內氏志賀菌抗原（天然提取物） | x1mg | Inactivated Shigella sonnei antigen (native extract) |
| MT-28PECU | 滅活的幽門螺桿菌抗原（天然提取物） | x1mg | Inactivated H. pylori antigen (native extract) |
| MT-29RVV | 滅活呼吸道合胞病毒抗原（天然提取物） | x1mg | Inactivated RSV antigen (native extract) |
| MT-28SPAU | 滅活沙門氏菌副傷寒A抗原（天然提取物） | x1mg | Inactivated Salmonella paratyphi A antigen (native extract) |
| MT-28SPBU | 滅活沙門氏菌副傷寒B抗原（天然提取物） | x1mg | Inactivated Salmonella paratyphi B antigen (native extract) |
| MT-28STU | 滅活傷寒沙門氏菌抗原（天然提取物） | x1mg | Inactivated Salmonella typhi antigen (native extract) |
| MT-28YE3U | 滅活小腸結腸炎耶爾森氏菌O:3抗原（天然提取物） | x1mg | Inactivated Yersinia enterocolitica O:3 antigen (native extract) |
| MT-28YE9U | 滅活小腸結腸炎耶爾森氏菌O:9抗原（天然提取物） | x1mg | Inactivated Yersinia enterocolitica O:9 antigen (native extract) |
| MT-29KOE | 滅活小球隱孢子蟲抗原（天然提取物） | x1mg | Inactivated Cryptosporidium parvum antigen (native extract) |
| MT-25EDP | 內阿米巴重組蛋白 | x1mg | Entamoeba dispar recombinant protein |
| MT-25NGI1 | 諾如病毒GI.1重組P結構域 | x1mg | Norovirus GI.1 recombinant P domain |
| MT-31NGA | 諾如病毒GI.1重組VLP | x1mg | Norovirus GI.1 recombinant VLP |
| MT-25NGI3 | 諾如病毒GI.3重組P結構域 | x1mg | Norovirus GI.3 recombinant P domain |
| MT-25NGII10 | 諾如病毒GII.10重組P結構域 | x1mg | Norovirus GII.10 recombinant P domain |
| MT-25NGII17 | 諾如病毒GII.17重組P結構域 | x1mg | Norovirus GII.17 recombinant P domain |
| MT-25NGII14 | 諾如病毒GII.4重組P結構域 | x1mg | Norovirus GII.4 recombinant P domain |
| MT-31NPA | 諾如病毒GII.4重組VLP | x1mg | Norovirus GII.4 recombinant VLP |
| MT-18NP8 | 諾如病毒GII單克隆抗體（克隆NP8） | x1mg | Anti-Norovirus GII Mab (clone NP8) |
| MT-18NG28 | 諾如病毒GI單克隆抗體（克隆NG28） | x1mg | Anti-Norovirus GI Mab (clone NG28) |
| MT-25HCP | 人類鈣衛蛋白重組蛋白 | x1mg | Human Calprotectin recombinant protein |
| MT-29HLF | 人乳鐵蛋白蛋白質（天然提取物） | x1mg | Human Lactoferrin protein (native extract) |
| MT-29HHB | 人血紅蛋白蛋白質（天然提取物） | x1mg | Human Haemoglobin protein (native extract) |
| MT-29HTF | 人轉鐵蛋白蛋白質（天然提取物） | x1mg | Human Transferrin protein (native extract) |
| MT-20TSS | 溶血性A鏈球菌抗體 | x1mg | Anti-Strep A Pab |
| MT-25EHP | 溶組織內阿米巴重組蛋白 | x1mg | Entamoeba histolytica recombinant protein |
| MT-16LC16 | 乳鐵蛋白單抗（克隆LC16） | x1mg | Anti-Lactoferrin Mab (clone LC16) |
| MT-16LC4 | 乳鐵蛋白單抗（克隆LC4） | x1mg | Anti-Lactoferrin Mab (clone LC4) |
| MT-18LN14 | 嗜肺軍團菌單抗(克隆LN14) | x1mg | Anti-Legionella pneumophila Mab (clone LN14) |
| MT-18LN29 | 嗜肺軍團菌單抗(克隆LN29) | x1mg | Anti-Legionella pneumophila Mab (clone LN29) |
| MT-16CA29 | 彎曲桿菌抗體（克隆ECA29） | x1mg | Anti-Campylobacter Mab (clone CA29) |
| MT-25CCP | 彎曲桿菌重組外膜蛋白 | x1mg | Campylobacter coli recombinant outer membrane protein |
| MT-25HEX | 腺病毒HEXON重組蛋白 | x1mg | Adenovirus HEXON recombinant protein |
| MT-18A14 | 腺病毒單克隆抗體（克隆A14） | x1mg | Anti-Adenovirus Mab (clone A14) |
| MT-18A15 | 腺病毒單克隆抗體（克隆A15） | x1mg | Anti-Adenovirus Mab (clone A15) |
| MT-18A15 | 腺病毒抗體（克隆A15） | x1mg | Anti-Adenovirus Mab (clone A15) |
| MT-25HEXR | 腺病毒六鄰體重組蛋白 | x1mg | Adenovirus HEXON recombinant protein |
| MT-18AT18 | 星狀病毒單克隆抗體（克隆AT18） | x1mg | Anti-Astrovirus Mab (clone AT18) |
| MT-18AT8 | 星狀病毒單克隆抗體（克隆AT8） | x1mg | Anti-Astrovirus Mab (clone AT8) |
| MT-25AST | 星狀病毒衣殼重組蛋白 | x1mg | Astrovirus capsid recombinant protein |
| MT-16F22 | 血紅蛋白單抗（克隆F22） | x1mg | Anti-Haemoglobin Mab (clone F22) |
| MT-18YB91 | 乙型流感病毒單抗（克隆YB91） | x1mg | Anti-Influenza B Mab (clone YB91) |
| MT-25FBN | 乙型流感病毒重組核蛋白 | x1mg | Influenza B recombinant nucleoprotein |
| MT-18K31 | 隱球菌抗體（克隆K31） | x1mg | Anti-Crypto Mab (clone K31) |
| MT-25PCH | 幽門螺桿菌重組外膜蛋白 | x1mg | H. pylori recombinant outer membrane protein |
| MT-16P2 | 幽門螺旋桿菌抗體（克隆P2）HP抗體 | x1mg | Anti-H. pylori Mab (clone P2) |
西班牙
我們的機體進化出了一些途徑來擺脫有缺陷的干細胞。來自科羅拉多大學癌癥中心的研究人員在發表于《干細胞》(Stem Cells)雜志上的一項研究中揭示，其中一條途徑就是通過“重編程”使得被輻射損傷的干細胞分化為不再能夠長久生存的其他細胞。輻射會使得干細胞喪失它的“干性”。這的確有意義：你不會希望受損的干細胞繼續留在這兒生成受損細胞。
這項研究還證實，當全身遭受輻射時清除輻射損傷干細胞的同一“編程”防衛機制導致了血癌生長(延伸閱讀：Sci Rep：福島核輻射后 動物血液出現異常 )。通過重編程這一防衛機制，我們或許能夠在全身輻射之后防止癌癥。
論文的資深作者、科羅拉多大學醫學院生物化學和分子遺傳學教授、癌癥中心研究員James DeGregori博士說：“機體并未進化至可以應對核反應堆泄漏及CT掃描。它只能一次處理少數接受危險劑量輻射或DNA遭到其他損傷的細胞。”
DeGregori、博士生Courtney Fleenor及同事們調查了全身輻射對于小鼠造血干細胞(HSCs)的影響。在這種情況下，輻射提高了造血干細胞系統中的細胞發生分化的幾率。只是，雖然大多數細胞會遵循這一指令，但有少數細胞則不會。攜帶一種特異突變的干細胞能夠違抗這一分化指令，保持它們的“干性”。遺傳抑制C/EBPα 可使得少數干細胞維持這一能力，繼續充當干細胞。在與其他細胞的競爭中，健康干細胞被清除，C/EBPα 減少的干細胞在血細胞生成系統中占據主導地位。通過這種方式，血液系統從C/EBPα +細胞轉變為主要是C/EBPα -細胞。
導致C/EBPα 基因受到抑制的一些突變和其他遺傳變異與人類急性髓性白血病有關聯。因此，并非是輻射所引起的突變，而是被錯誤干細胞重建的血液系統引起了經歷過輻射的人們的癌癥風險。
DeGregori說：“這是由自然選擇所驅動的進化。在健康血液系統中，健康干細胞會在競爭中勝過具有C/EBPα 突變的干細胞。但當輻射降低干細胞群的健康和適應性時，一直在那的突變細胞突然得到了接管的機會。”
從花栗鼠和松鼠的角度來思考這一問題：減少生物系統中的花栗鼠種群或許可使得松鼠興旺——尤其如果是減少花栗鼠的途經改變了生態系統從而有利于松鼠，這與輻射改變機體來支持C/EBPα 突變干細胞相類似。
西班牙
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【公司名稱】 廣州健侖生物科技有限公司
【市場部】 楊永漢
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【騰訊 】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室
Our body has evolved ways to get rid of defective stem cells. In a study published in the journal Stem Cells, researchers at the University of Colorado Cancer Center revealed that one of the ways to do this is through reprogramming to differentiate irradiated stem cells that no longer survive Other cells. Radiation can cause stem cells to lose their "dryness." This does make sense: You do not want damaged stem cells to stay there to create damaged cells.
The study also confirmed that the same "programmed" defense mechanism that cleared radiation-damaged stem cells when exposed to radiation throughout the body led to the growth of blood cancers (Extended reading: Sci Rep: abnormal blood flow in animals after Fukushima nuclear irradiation). By reprogramming this defense mechanism, we may be able to prevent cancer after systemic radiation.
"The body has not evolved to handle nuclear reactor leaks and CT scans," said senior author of the paper, Dr. James DeGregori, a professor of biochemistry and molecular genetics at the University of Colorado School of Medicine and a researcher at the Cancer Center. "It can only deal with a small number of patients receiving radiation exposure at a dangerous dose or The DNA was damaged by other cells. "
DeGregori, PhD candidate Courtney Fleenor and colleagues investigated the effects of whole body radiation on mouse hematopoietic stem cells (HSCs). In this case, radiation increases the chance of cell differentiation in the hematopoietic stem cell system. Only, although most cells follow this instruction, a few cells do not. Stem cells that carry a specific mutation are able to defy this differentiation and keep them "dry". Genetic inhibition of C / EBPα allows a small number of stem cells to maintain this capacity and continue to act as stem cells. In competition with other cells, healthy stem cells are cleared and stem cells with reduced C / EBPα dominate the hematopoietic system. In this way, the blood system changes from C / EBPα + cells to predominantly C / EBPα - cells.
Some mutations and other genetic variations that lead to the inhibition of the C / EBPα gene have been linked to human acute myeloid leukemia. Therefore, rather than the mutations caused by radiation, the blood system reconstructed by the wrong stem cells raises the cancer risk for those who have experienced radiation.
"This is evolution driven by natural selection," said DeGregori. "In a healthy blood system, healthy stem cells outperform stem cells with C / EBPα mutations in competition, but when radiation reduces the health and fitness of the stem cell population, Suddenly the mutant cells there got the chance to take over. "
Consider this from the chipmunk and squirrel perspectives: Reducing the population of chipmunk in the biological system may allow squirrels to thrive - especially if the path to the chipmunk is diminished by altering the ecosystem to benefit the squirrel, To support similar C / EBPα mutant stem cells.
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