美國Seracare大腸桿菌O157:H7陽性對照

廣州健侖生物科技有限公司

廣州健侖長期供應各種生物原料，主要代理品牌：美國Seracare、西班牙Certest、美國Fuller等等。

主要產品包括各種標準品、陽性對照品、陽性質控品、單克隆抗原抗體。

其中常見的有：弓形蟲病、西尼羅河病毒、類風濕因子、瘧疾、麻疹、萊姆病、百日咳桿菌、大腸桿菌、鼠傷寒沙門氏菌、李斯特菌等陽性對照品。

美國Seracare大腸桿菌O157:H7陽性對照

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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此研究主題刊登于2014年8月的《實驗生物及醫學》，證明了丙酮酸脫氫酶復合體不僅對于胰島素基因表達和葡萄糖刺激的胰島素分泌非常必要，同時還直接影響β-細胞的生長和成熟，更闡明了葡萄糖代謝會直接調控β細胞質量和可塑性。
紐約州立大學水牛城分校生物化學、醫學和生物醫學科學學院的特聘教授，同時也是資深作者的莫成?帕特爾博士說道：“這些發現顯示了葡萄糖代謝是調控β細胞質量的主要調控因子，很可能是獨立的訊息傳遞路徑，例如可能不同于胰島素受體作用物2訊息傳遞路徑。”
《實驗生物及醫學》期刊主編史蒂芬?古德曼博士說：“帕特等人的研究利用小鼠基因剔除的方式來研究丙酮酸脫氫酶復合體在胰臟β-細胞發育所扮演的角色。他們的研究證明丙酮酸脫氫酶復合體直接影響β-細胞的細胞大小及可塑性。”
盧森堡大學盧森堡中心系統生物醫學研究院(LCSB)的科學家們已經將皮膚細胞神經元重編程植入到小鼠大腦并*維持長期穩定。植入后6個月，神經元功能上*整合到大腦。這一成功，持久穩定，神經元的植入為將來治療帕金森病人智力時，用健康的神經元替換患病的神經元增加希望。
圖片顯示大腦切片部分，其中被移植的誘導神經干細胞*整合到大腦的神經元網絡(藍色)發育成一個復雜的功能性神經元。
LCSB的測試中，馬克斯普朗克研究所和大學醫院明斯特和比勒費爾德大學的研究小組和同事成功將由皮膚細胞進行重編程誘導的神經元在小鼠大腦中創建了穩定的神經組織。產生神經元的干細胞研究者的技術，或者更具體的說誘導神經元干細胞(iNSC)，在培養皿中培養的宿主皮膚細胞在很大程度上改善了植入細胞的相容性。
治療的小鼠沒有顯示出不利的副作用，甚至在植入六個月后成為大腦的海馬和皮質區域。植入神經元*整合到大腦的復雜網絡。該神經元表現出正常活性并且通過新形成的突觸——神經細胞之間的接觸點連接到原始腦細胞。
狡猾的寄生蟲在其生命周期中常常經歷顯著的外形變化，讓它們能夠適應不同的生活環境并且繁盛起來。但是發表在《細胞生物學雜志》(The Journal of Cell biology)的一項研究發現，這些變形看上去可能沒有看上去的那么困難。
非洲的“昏睡病”是一種由稱為布氏錐蟲(Trypanosoma brucei)的由采采蠅傳播的寄生蟲物種導致的疾病。這種單細胞寄生蟲擁有一個內部儲藏著這個細胞的線粒體DNA的動基體以及對于細胞運動具有關鍵作用的一個伸出的鞭毛。布氏錐蟲在其發育周期經歷了外形和構成的重大變化。在一個稱為錐鞭毛體(trypomastigote)的階段，動基體位于核之后，而幾乎鞭毛的所有部分都與細胞相連。另一方面，在短膜蟲期(epimastigote)階段，動基體在核之前，而且只有鞭毛的一部分與細胞相連。布氏錐蟲的近親有許多不同的形狀，這表明這些寄生蟲在進化中也改變了它們的形態。

美國Seracare

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、食品安全、化妝品檢測、藥物濫用檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

The subject of the study, published in August 2014, "Experimental Biology and Medicine," demonstrates that pyruvate dehydrogenase complexes are essential not only for insulin gene expression and glucose-stimulated insulin secretion but also for β-cell growth and Mature, but also shows that glucose metabolism will directly regulate β-cell quality and plasticity.
Dr. Moblie Pa, Distinguished Professor of Biochemistry, Medical and Biomedical Sciences at the State University of New York at Buffalo, said: "These findings show that glucose metabolism is a major regulator of beta-cell mass, Probably an independent messaging pathway, for example, may differ from the insulin receptor 2 signaling pathway. "
Dr. Stephen Goodman, editor-in-chief of Experimental Biology and Medicine, said: "Pat et al's study explored the role of the pyruvate dehydrogenase complex in the pancreatic β-cell development using gene knockout in mice. Their research shows that pyruvate dehydrogenase complex directly affects the cell size and plasticity of β-cells. "
Scientists at the Luxembourg Center for System Biomedicine Research (LCSB) at the University of Luxembourg have already implanted skin-cell neurons into the mouse brains and have maintained long-term stability for the first time. Six months after implantation, neurons functionally integrate compley into the brain. This success, lasting stability, neuronal implantation for the future treatment of Parkinson's patients inligence, with healthy neurons to replace diseased neurons increase hope.
The picture shows a section of the brain that grows into a complex functional neuron in which the transplanted neural stem cells are fully integrated into the brain's neuronal network (blue).
In the latest LCSB test, research groups and colleagues at the Max Planck University and the University Hospital in Münster and Bielefeld succeeded in creating neurons that are reprogrammed by skin cells to create stable Nerve tissue. Stem Cells Producing Neurons Researchers' technology, or more specifically, induction of neuronal stem cells (iNSCs), host skin cells cultured in petri dishes greatly improve the compatibility of the implanted cells.
The treated mice showed no adverse side effects and became brain regions of the hippocampus and cortex even six months after implantation. Implanted neurons are fully integrated into the complex network of the brain. The neurons exhibit normal activity and are connected to the naive brain cells through the contact points between the newly formed synaptic-neurons.
Cunning parasites often experience significant changes in shape throughout their life cycle, allowing them to adapt to different living environments and flourish. But a study published in The Journal of Cell biology found that these distortions may not seem so difficult.
Sleeping sickness in Africa is a disease caused by a parasitic species transmitted by tsetse flies called Trypanosoma brucei. The unicellular parasite has a moving body that holds the mitochondrial DNA of the cell inside, and a protruding flagella that is crucial for cell motility. Trypanosoma brucei undergoes significant changes in shape and composition during its developmental cycle. In a phase called trypomastigote, the motile matrix is ??behind the nucleus, with almost all parts of the flagellum connected to the cell. On the other hand, in the epimastigote stage, the motile matrix precedes the nucleus, and only a portion of the flagella is attached to the cell. The relatives of Trypanosoma brucei have many different shapes, suggesting that these parasites also changed their morphology during evolution.