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-試劑盒
廣州健侖生物科技?有限公司
本司長(zhǎng)期供應(yīng)軍團(tuán)菌試劑盒 其主要品牌包括美國(guó)NovaBios、廣州健侖、廣州創(chuàng)侖等進(jìn)口產(chǎn)品,國(guó)產(chǎn)產(chǎn)品,試劑盒的實(shí)驗(yàn)方法是膠體金方法。
我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。
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【包裝規(guī)格】25T/盒
名稱 | 規(guī)格 | 單位 | 備注 | 貨期 | 產(chǎn)地或品牌 | |
軍團(tuán)菌診斷血清 (貨號(hào):294623) | 2ml×10種 7-15群 共19瓶 | 套 | 包含:嗜肺性軍團(tuán)菌1~6群、波茲曼軍團(tuán)菌、杜莫氏軍團(tuán)菌、高爾曼軍團(tuán)菌、麥克答德軍團(tuán)菌診斷血清 | 6-8周 | 日本生研 | |
嗜肺軍團(tuán)菌核酸 熒光PCR檢測(cè)試劑盒 | 25T/盒 | 盒 | *適用于 Ⅰ、Ⅱ、Ⅲ、Ⅳ類儀器 | 5天 | 創(chuàng)侖生物制品 | |
嗜肺軍團(tuán)菌(1—7)IgG試劑盒 | 96T/盒 | 盒 | ELISA定量 | 30天 | 健侖 | |
嗜肺軍團(tuán)菌(1—8)IgM試劑盒 | 96T/盒 | 盒 | ELISA定量 | |||
軍團(tuán)菌CYE瓊脂基礎(chǔ) | 500g | 瓶 | 用于軍團(tuán)菌培養(yǎng) | 30-40天 | 英國(guó)OXOID | |
BCYE培養(yǎng)基 (檢測(cè)軍團(tuán)菌的 標(biāo)準(zhǔn)培養(yǎng)基) | BCYE添加劑 | 10小瓶/套 | 套 | 每瓶添加100ml培養(yǎng)基 | ||
不含半胱氨酸的 BCYE添加劑 | 10小瓶/套 | 套 | 每瓶添加100ml培養(yǎng)基 | |||
BMPA 選擇培養(yǎng)基 | 軍團(tuán)菌 BMPA添加劑 | 10小瓶/套 | 套 | 每瓶添加100ml培養(yǎng)基 | ||
GVPC 選擇培養(yǎng)基 | 軍團(tuán)菌 GVPC添加劑 | 10小瓶/套 | 套 | 每瓶添加500ml培養(yǎng)基 | ||
MWY 選擇培養(yǎng)基 | 軍團(tuán)菌 MWY添加劑 | 10小瓶/套 | 套 | 每瓶添加100ml培養(yǎng)基 |
【研發(fā)原理】
軍團(tuán)菌是近年來(lái)發(fā)現(xiàn)的新型病原菌,它侵入人體后主要引起軍團(tuán)菌肺炎。據(jù)發(fā)達(dá)國(guó)家報(bào)道,軍團(tuán)菌肺炎占肺部感染總數(shù)的7%~10%,老年人中會(huì)更高。該病發(fā)病急、死亡率高,因此臨床上迫切需要能夠快速準(zhǔn)確檢測(cè)軍團(tuán)菌的方法。根據(jù)市場(chǎng)需要,我公司攻克多項(xiàng)技術(shù)難關(guān),成功研制出軍團(tuán)菌抗體快速金標(biāo)記檢定卡。
【產(chǎn)品特點(diǎn)】
★操作簡(jiǎn)便,無(wú)需其它儀器和試劑,易于在各級(jí)醫(yī)院推廣;
★反應(yīng)迅速,5分鐘內(nèi)即可得到結(jié)果;
★結(jié)果清晰,易于判定;
★敏感度高,特異性強(qiáng)。
游泳池預(yù)防軍團(tuán)菌檢測(cè)試紙
【檢測(cè)方法】
1)從鋁袋中取出所需數(shù)量的磁帶,并將它們放在水平面上。
2)使用定量移液器慢慢地吸取90μL或更多的樣品。 通過(guò)一次移液操作,將全部樣品滴入盒子的樣品位置。 滴下后,剩余的標(biāo)本留在吸管內(nèi)。
3)在室溫(15-30°C)下將盒子放置15分鐘,
4)檢查盒子評(píng)估地點(diǎn)是否有線(標(biāo)有“C”和“T”)。
結(jié)果
應(yīng)該在反應(yīng)開(kāi)始15分鐘后及時(shí)進(jìn)行評(píng)估。 檢查評(píng)估地點(diǎn),并通過(guò)下面描述的紅線來(lái)評(píng)估結(jié)果。
1)正面:紅色控制線和測(cè)試線均在評(píng)估現(xiàn)場(chǎng)。 當(dāng)兩條線在反應(yīng)時(shí)間結(jié)束之前出現(xiàn)時(shí)結(jié)果是肯定的。
2)否定的:評(píng)估地點(diǎn)只有一條紅色的控制線。
3)無(wú)效:無(wú)論控制線是否存在,無(wú)論是否存在測(cè)試線,測(cè)試均無(wú)效。
想了解更多的韓國(guó)SD產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼:我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。
二維碼掃一掃
【公司名稱】 廣州健侖生物科技有限公司
【】 楊永漢
【】
【騰訊 】
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-3室
【企業(yè)文化宣傳】
Spread of body lice is the main medium of transmission of the disease, head lice and pubic lice may also be used as a medium, but of little significance. Tick ??spread mainly between animals Plagaris rickettsia, whether it can spread in people for further study. Body lice inhale blood, active in the appropriate temperature, easy to spread in the crowd, when the patient quickly fled to find the new owner. Rodents of the subject's louse do not contain rickettsia in the saliva, but excretion of the pathogen-containing manure on the skin while sucking the human blood can cause the rickettsia to enter the body through the punctures or scratches. Sometimes people scratch the tick and the lice crushed, then the lice body pathogens can also catch bacteria and inoculated in the skin. The pathogens in the louse droppings can be aerosols and inhaled into the respiratory tract, or enter the body through the conjunctiva infection occurs, laboratory workers prone to aerosol infection. There are people who have a bad habit of lice crushing, which can be infected by rickettsia through the oral mucosa. There are very few chances of being attacked by autopsy or blood transfusion. Louse sucking the patient's blood pathogen into the gastrointestinal tract, rickettsia that invade lice intestinal epithelial cells, 4 to 5 days after the cell swelling due to rupture, so a large number of rickettsial enter the intestine, and with lice excrement Excreted. Generally 7 to 10 days after infection can be extended to more than 3 weeks, the louse itself also died of intestinal obstruction caused by infection. Rickettsia in lice do not pass through eggs. Lice from fever patients and deaths and healthy people, which is conducive to the spread of the disease. So far, the transmission mode of "human-lice-human" with the patient as the source of infection and body lice as the transmission medium is still the basic concept of epidemiology of this disease.
(C) population susceptibility to the disease in all age groups are highly susceptible, children under 15 years of age when the disease was mild. According to reports from some areas in China, it is not uncommon for light or atypical cases to have a fairly long-lasting immunity after getting sick and even have the possibility of being infected again. In addition to recurrent typhus, relapse (short term) is extremely rare.
(D) epidemiological characteristics of the epidemic and human lice are closely related. The prevalence of the disease in winter and spring are more common, because of the cold climate, clothing thick, and less change wash, it is conducive to parasitic and reproductive lice. The disease occurred in the cold more in the past, but in recent years, tropical Africa such as more cases have been reported. Bad weather, famine and sanitation can easily lead to epidemics. The company is located in:
[Pathogenesis and pathological changes]
Rickettsia invades the human body, the first breed in small vascular endothelial cells, rupture of cells rupture into the blood to form rickethaliosis, invasion of systemic small vascular endothelial cells. Pathogen death, the release of a large number of toxins can cause symptoms of systemic poisoning. The second week of the course of the body with the emergence of anti-infective immune response to bacterial pathogens, the vascular lesions further aggravated.
Pathological changes are characterized by proliferative, thrombotic, necrotizing vasculitis and perivascular inflammatory cell infiltration formed by typhus nodules. This proliferation of thrombotic necrotizing vasculitis can be distributed throughout the body tissues and organs. More common in the skin, myocardium, central nervous system. The central nervous system to the cerebral cortex, medulla oblongata, basal ganglia most severely damaged, pons, followed by the spinal cord. Meninges can be acute serous inflammation. The lungs may have interstitial inflammation and bronchopneumonia. Hepatic portal area with basophilic mononuclear cell infiltration, liver cells have varying degrees of fatty degeneration and focal necrosis and mononuclear cell infiltration. The main renal interstitial inflammatory lesions.
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