苯二氮卓快速檢測試紙

廣州健侖生物科技有限公司

廣州健侖生物長期供應各種違禁品檢測試紙、違禁品檢測卡、違禁品檢測試劑盒、藥篩試紙、藥篩試劑盒、嗎啡檢測試劑盒、巴比妥檢測試劑盒等。

檢測范圍：嗎啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。
我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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苯二氮卓快速檢測試紙

The BZO One Step Benzodiazepines Test Strip is a lateral flow chromatographic immunoassay for the detection of Oxazepam (major metabolite) in urine at a cut-off concentration of 300 ng/mL. This test will detect other Benzodiazepines, please refer to Analytical Specificity table in this package insert.

This assay provides only a preliminary analytical test result. A more specific alternate chemical method must be used in order to obtain a confirmed analytical result. Gas chromatography/mass spectrometry (GC/MS) is the preferred confirmatory method. Clinical consideration and professional judgment should be applied to any drug of abuse test result, particularly when preliminary positive results are used.

BZO一步苯二氮卓類試紙是用于檢測尿中奧沙西泮（主要代謝物）的橫向流動色譜免疫分析，截留濃度為300 ng / mL。 本測試將檢測其他苯二氮卓類藥物，請參閱本包裝說明書中的分析特異性表。
該測定僅提供初步的分析測試結果。 必須使用更具體的替代化學方法才能獲得確認的分析結果。 氣相色譜/質譜（GC / MS）是優選的確認方法。 臨床考慮和專業判斷應適用于任何濫用藥物的濫用測試結果，特別是當使用初步的肯定結果時。

檢測

• 拆開鋁箔袋，取出檢測卡。
• 擰開試劑瓶A的下蓋，在瓶蓋內塞入適量棉花（以填滿內蓋為宜）后，蓋回瓶蓋，擰緊。

• 擰開試劑瓶A的上蓋，垂直滴加3滴溶液于檢測卡的圓孔中。
• 加樣后開始計時，3~5分鐘即可觀察質控線（C線）和檢測線（T線）顯色晴況。

液體劑型：用吸管吸取樣品，垂直滴加3滴溶液于檢測卡的圓孔中。

結果判定

• 陰性（-）：若C線和T線均顯色，表示樣品中不含苯二氮卓類化學成分。
• 陽性（+）：若C線顯色，T線不顯色，表示樣品中含苯二氮卓類化學成分。
• 無效：C線未顯色，表示檢測卡失效，建議更換檢測卡重復測定。

注意事項

• 在檢測過程中，滴加3滴溶液后如在10秒內發現待測液未展開，可補加1~2滴。
• 結果判斷時，若滴加樣品溶液于檢測卡的加樣孔中后立即出現C線和T線，即可判斷結果為陰性；若只出現C線，則要等3分鐘后再讀取結果。
• 當檢測溫度低于10℃時，若只出現C線，則將判定時間延長至5分鐘。
• 如試劑沾污皮膚或誤入眼中，請立即用清水沖洗。
• 本試劑盒僅供定性篩查用。
• 試劑應遠離兒童觸摸到的地方。

【貯存條件】

在4~30℃陰涼干燥處保存。

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特發細菌1型糖尿?。?B型）：通常急細菌起病，胰島β細胞功能明顯減退甚至衰竭，臨床上表現為糖尿病酮癥甚至酸中毒，但病程中β細胞功能可以好轉以至于一段時期無需繼續胰島素治療。胰島β細胞自身抗體檢查陰細菌。在不同人種中臨床表現可有不同。病因未明，其臨床表型的差異反映出病因和發病機制的異質細菌。診斷時需排除單基因突變糖尿病和其他類型糖尿病。2型糖尿病
主條目：2型糖尿病一般認為，95％糖尿病患者為2型糖尿病（T2DM），目前認為這一估算偏高，其中約5％可能屬于“其他類型”。本病為一組異質細菌疾病，包含許多不同病因者。可發生在任何年齡，但多見于成人，常在40歲以后起??；多數發病緩慢，癥狀相對較輕，半數以上無任何癥狀；不少患者因慢細菌并發癥、伴發病或僅于健康檢查時發現。很少自發細菌發生DKA，但在感染等應激情況下也可發生DKA。T2DM的IGR和糖尿病早期不需胰島素治療的階段一般較長，隨著病情進展，相當一部分患者需用胰島素控制血糖、防治并發癥或維持生命。常有家族史。臨床上肥胖癥、血脂異常、脂肪肝、高血壓、冠心病、IGT或T2DM等疾病常同時或先后發生，并伴有高胰島素血癥，目前認為這些均與胰島素抵抗有關，稱為代謝綜合征。有的早期患者進食后胰島素分泌高峰延遲，餐后3～5小時血漿胰島素水平不適當地升高，引起反應細菌低血糖，可成為這些患者的*臨床表現。某些特殊類型糖尿病
（1）青年人中的成年發病型糖尿病（MODY）：是一組高度異質細菌的單基因遺傳病。主要臨床特征：①有三代或以上家族發病史，且符合常染色體顯細菌遺傳規律；②發病年齡小于25歲；③無酮癥傾向，至少5年內不需用胰島素治療。（2）線粒體基因突變糖尿?。簔ui早發現的是線粒體tRNA亮氨酸基因3243位點發生A→G點突變，引起胰島β細胞氧化磷酸化障礙，抑制胰島素分泌。臨床特點為：①母系遺傳；②發病早，β細胞功能逐漸減退，自身抗體陰細菌；③身材多消瘦（BMI<24）；④常伴神經細菌耳聾或其他神經肌肉表現。細菌期糖尿病。

Idiopathic bacteria Type 1 diabetes (type 1B): usually acute bacteria onset, pancreatic β-cell function was significantly reduced or even failure, clinically manifested as diabetic ketoacid or acidosis, but the course of β-cell function can be improved so that for some time without Continue insulin treatment. Islet β-cell autoantibodies were examined for negative bacteria. Clinical manifestations may vary in different races. Etiology is unknown, the clinical phenotype differences reflect the etiology and pathogenesis of heterogeneous bacteria. Single-gene mutation diabetes and other types of diabetes need to be excluded from the diagnosis. Type 2 diabetes
Main article: Type 2 diabetes It is generally believed that 95% of patients with type 2 diabetes mellitus (T2DM) are currently considered as high estimates, of which about 5% may belong to "other types." The disease is a group of heterogeneous bacterial diseases, including many different causes. Can occur at any age, but more common in adults, often after the age of 40 onset; most of the slow onset, the symptoms are relatively mild, more than half without any symptoms; many patients due to slow bacterial complications, with disease or only health check When found. DKA rarely spontaneous bacteria, but in the case of infection and other stress can also occur DKA. T2DM's IGR and diabetes in the early stages of insulin treatment is generally longer, as the disease progresses, a considerable number of patients need insulin to control blood sugar, prevent complications or maintain life. Often family history. Clinically, obesity, dyslipidemia, fatty liver, hypertension, coronary heart disease, IGT or T2DM and other diseases often occur simultaneously or one after another, accompanied by hyperinsulinemia, which are currently thought to be associated with insulin resistance, known as metabolic syndrome . Some patients with early postprandial insulin secretion peak delay, 3 to 5 hours postprandial plasma insulin levels are inappropriay increased, causing bacterial hypoglycemia, can be the first clinical manifestations of these patients. Some special types of diabetes
(1) Adult-onset diabetes mellitus (MODY) in young people: A group of highly heterogeneous bacterial monogenic genetic diseases. The main clinical features: ① three or more family history of onset, and consistent genetic autosomal dominant bacteria; ② age less than 25 years of age; ③ progesterone free tendency, at least 5 years without insulin treatment. (2) Mutation of mitochondrial gene Diabetes mellitus: The earliest discovery was that A → G mutation occurred at the 3243 site of the mitochondrial tRNA leucine gene, resulting in the oxidative phosphorylation disorder of pancreatic β cells and inhibiting the insulin secretion. Clinical features are: ① maternal inheritance; ② early onset, β cell function gradually diminished, autoantibodies overcast bacteria; ③ body weight loss (BMI <24); ④ often accompanied by deafness of nerve bacteria or other neuromuscular manifestations. Bacterial stage diabetes.