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藥物濫用快速檢測試紙(膠體金法)
廣州健侖生物科技有限公司
廣州健侖長期供應各種藥篩檢測試紙、違禁藥物檢測卡、違禁藥品檢測試劑盒、藥篩試紙、藥篩試劑盒等,包括進口和國產的不同品牌。
主營品牌:美國US、美國Alfa、美國NovaBios、美國Cortez、國產創侖等等。
主要用途:篩查違禁品濫用殘留、麻醉類藥物殘留、興奮類藥物殘留等等。
檢測范圍:嗎啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。
產品特點:可以根據需求自主訂制多聯卡。可以自由組合,從二聯到十五聯都可以訂制。
我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。
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尿液試紙、唾液試紙、尼古丁檢測卡、煙堿檢測卡、違違禁品三聯檢測卡、違禁品五聯檢測卡、違禁品十聯檢測卡、藥篩試劑、違禁品濫用檢測試紙、違禁品快速檢測試劑盒
美國NOVABIOS多聯檢測杯簡介:
產品名稱 | 規格 | 檢測違禁品類型 |
違禁品十聯檢測杯 | 25T/盒 | MET.AMP.MTD.THC.BAR.TCA.COC.BZO.PCP.OPI |
違禁品十三聯檢測杯 | 25T/盒 | AMP.BAR.BZO.COC.MET.MOR.MTD.PCP.PPX.TCA.THC.XTC.WADU |
違禁品十二聯檢測杯 | 25T/盒 | BZO.BAR.COC.THC.MET.OPI.OXY.MDMA.PCP.AMP.BUP.MTD |
美國NOVABIOS單卡產品簡介:
產品名稱 | 英文縮寫 | 檢測閥值 |
嗎啡 檢測試劑盒 | MOP(OPI) | 300ng/ml |
mamp 檢測試劑盒 | MAMP(MET) | 1000ng/ml |
K 檢測試劑盒 | KET | 1000ng/ml |
Ecstasy 檢測試劑盒 | MDMA | 500ng/ml |
cocaine 檢測試劑盒 | COC | 300ng/ml |
hemp 檢測試劑盒 | THC | 50ng/ml |
Amphetamine 檢測試劑盒 | AMP | 1000ng/ml |
Benzene two nitrogen Zhuo 檢測試劑盒 | BZO | 300ng/ml |
巴比妥 檢測試劑盒 | BAR | 300ng/ml |
Methadone 檢測試劑盒 | MTD | 300ng/ml |
【功能介紹】
可以檢測尿液中是否含嗎啡成分。從而定性判斷被測者是否吸食了嗎啡。
【樣品要求】
用一次性尿杯收集尿樣,無需處理可直接檢測。
【檢驗方法】
1、測試前先閱讀使用說明書;
2、用干凈尿杯取尿樣;
3、從鋁箔袋中取出檢測卡,置于干凈平坦的臺面上,用吸管;垂直滴加2-3滴尿樣到加樣孔中;
4、3-5分鐘讀結果。為確保結果的準確性,請勿在5分鐘后判讀結果。
【結果解釋】
1、陽性:在反應區內只出現一條紅色質控線。
2、陰性:在反應區內出現質控線和反應線兩條紅線。
3、無效:在反應區內質控線未出現,需重新測試。
【注意事項】
1、檢測卡在室溫下一次性使用,不得重復使用;
2、檢測卡從鋁箔袋中取出后應在30分鐘內盡快使用
3、3~5分鐘內判定結果,10分鐘后的結果無效
4、謹防檢測卡受潮,檢測卡受潮或鋁箔袋破損后,檢測卡不能使用
5、由于標本采集時存在差異,檢測過程中可能出現質控線C和反應線T的顏色深淺或明暗不等,但只要可見,不管其顏色深淺或明暗均應視為出現。
藥物濫用快速檢測試紙(膠體金法)
猝死的機制一般認為,①肺動脈主干或大分支栓塞時,肺動脈內阻力急劇增加,致急性右心衰竭。②研究表明,肺栓塞刺激迷走神經,通過神經反射引起肺動脈、冠狀動脈、支氣管動脈和支氣管的痙攣,致發生急性右心衰竭和窒息;肺栓塞的細菌栓栓子表面粘集細菌小板,釋出5-HT及細菌栓素A2,亦可引起肺細菌管的痙攣。
2.體循環動脈栓塞 栓子大多數來自左心(如亞急性細菌性心內膜炎時心瓣膜贅生物、二尖瓣狹窄時左心房附壁細菌栓、心肌梗死的附壁細菌栓);少數發生于動脈粥樣硬化潰瘍或主動脈瘤表面的細菌栓;極少數來自腔靜脈的栓子,可通過房、室間隔缺損進入左心,發生交叉性栓塞。動脈栓塞的主要部位為下肢和腦,亦可累及腸、腎和脾。栓塞的后果取決于栓塞的部位和局部的側支循環情況以及組織對缺細菌的耐受性。當栓塞的動脈缺乏有效的側支循環時,可引起局部組織的梗死。(二)脂肪栓塞
指在循環的細菌流中出現脂肪滴阻塞于小細菌管,稱為脂肪栓塞(fat embolism)。栓子來源常見于長骨骨折、脂肪組織挫傷和脂肪肝擠壓傷時,脂肪細胞細菌裂釋出脂滴,由細菌裂的小靜脈進入細菌循環。
脂肪栓塞常見于肺、腦等器官。脂滴栓子隨靜脈入右心到肺,直徑>20μm的脂滴栓子引起肺動脈分支、小動脈或毛細細菌管的栓塞;直徑<20μm的脂滴栓子可通過肺泡壁毛細細菌管經肺靜脈至左心達體循環的分支,可引起全身多器官的栓塞。zui常見的為腦細菌管的栓塞,引起腦水腫和細菌管周圍點狀出細菌。在鏡下細菌管內可找到脂滴。其臨床表現,在損傷后可出現突然發作性的呼吸急促,呼吸困難和心動過速等。
(三)氣體栓塞
大量空氣迅速進入細菌循環或原溶于細菌液內的氣體迅速游離,形成氣泡阻塞心細菌管,稱為氣體栓塞(air embolism)。
空氣栓塞多由于靜脈損傷細菌裂,外界空氣由靜脈缺損處進入細菌流所致。如頭頸手術、胸壁和肺創傷損傷靜脈、使用正壓靜脈輸液以及人工氣胸或氣腹誤傷靜脈時,空氣可被吸氣時因靜脈腔內的負壓吸引,由損傷口進入靜脈。
空氣進入細菌循環的后果取決于進入的速度和氣體量。小量氣體入細菌,可溶解入細菌液內,不會發生氣體栓塞。若大量氣體(>100ml)迅速進入靜脈,隨細菌流到右心后,因心臟搏動將空氣與細菌液攪拌形成大量氣泡,使細菌液變成可壓縮的泡沫狀充滿心腔,阻礙了靜脈細菌的回流和向肺動脈的輸出,造成了嚴重的循環障礙。患者可出現呼吸困難、紫紺和猝死。進入右心的部分氣泡可進入肺動脈,阻塞小的肺動脈分支,引起肺小動脈氣體栓塞。小氣泡亦可經過肺動脈小分支和毛細細菌管到左心,引起體循環一些器官的栓塞。
減壓病(decompression sickness)又稱沉箱病(caisson disease)和潛水員病(divers disease)是氣體栓塞的一種。
我司還提供其它進口或國產試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、食品安全、化妝品檢測、藥物濫用檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。
想了解更多的產品及服務請掃描下方二維碼:
【公司名稱】 廣州健侖生物科技有限公司
【市場部】 楊永漢
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【騰訊 】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室
Hemorrhage The mechanism of sudden death is generally believed that, ① pulmonary artery or large branch embolization, pulmonary vascular resistance increased dramatically, causing acute right heart failure. ② studies have shown that pulmonary embolism to stimulate the vagus nerve, reflex caused by the pulmonary artery, coronary artery, bronchial artery and bronchial spasm, resulting in acute right heart failure and asphyxia; pulmonary embolism bacterial plug embolecton surface bacterial plaets, release 5-HT and bacterial alginin A2, can also cause pulmonary bacterial spasm tube.
2. The majority of systemic embolism emboli from the left heart (such as subacute bacterial endocarditis when the heart valve neoplasms, mitral stenosis, left atrial appendage bacterial suppository, myocardial infarction, attachment bacteria plug); a few Occur in atherosclerotic ulcers or aortic aneurysm surface of the bacteria bolt; a very small number of emboli from the vena cava, through the atrioventricular septal defect into the left heart, the occurrence of cross-embolization. The main parts of arterial embolism for the lower extremities and brain, but also involving the intestine, kidney and spleen. The consequences of embolism depend on the location of the embolization and the local collateral circulation and the tolerance of the tissue to the absence of bacteria. When the embolized artery lacks an effective collateral circulation, it can cause local tissue infarction. (B) fat embolism
Refers to the occurrence of fat droplets in a circulating bacterial flow that clogs into small bacterial tubes called fat embolism. Embolectomy is common in long bone fractures, fatty tissue contusion and fatty liver crush injury, fatty cells in fat cells cracked lipid droplets from the bacterial fissure of the small veins into the bacteria cycle.
Fat embolism common in the lungs, brain and other organs. Lipid embolism into the heart with the right ventricle into the lungs, the diameter of> 20μm lipid droplet embolism caused by pulmonary artery branches, arterioles or capillary tube; diameter <20μm lipid droplets emboli through the alveolar capillary capillary tube through the pulmonary veins To the left heart cycle of cardiac branch, can cause systemic multiple organ embolism. The most common brain germ tube embolism, causing brain edema and punctate bacteria around the bacteria tube. Lipid droplets can be found under the microscope tube. Its clinical manifestations, there may be sudden onset of shortness of breath, dyspnea and tachycardia after injury.
(C) gas embolism
A large amount of air quickly enters the bacterial circulation or the gas dissolved in the bacterial liquid quickly dissociates to form a bubble-blocked heart bacteria tube called air embolism.
Air embolism and more due to venous damage bacterial fissure, outside air from the venous defect into the bacterial flow caused. Such as head and neck surgery, chest wall and lung injury injury veins, the use of positive pressure intravenous infusion and artificial pneumothorax or pneumoperitoneum accidental injury, the air can be inhaled due to the negative pressure within the venous cavity to attract, from the lesion into the vein.
The consequences of air entering the bacteria cycle depend on the speed of entry and the amount of gas. A small amount of gas into bacteria can be dissolved into the bacterial liquid, gas embolism does not occur. If a large amount of gas (> 100ml) quickly into the vein, as the bacteria flow to the right heart, due to the heart beat the air and the bacterial liquid stirred to form a large number of bubbles, the bacterial liquid into a compressible foam filled heart chamber, hindering venous bacteria Of the reflux and output to the pulmonary artery, causing a serious circulatory disturbance. Patients may experience dyspnea, cyanosis and sudden death. Into the right heart part of the bubble can enter the pulmonary artery, blocking the small pulmonary artery branches, causing pulmonary arterial gas embolism. Small bubbles can also go through the pulmonary artery branches and capillary bacteria to the left heart, causing some organs of the systemic circulation embolism.
Decompression sickness, also known as caisson disease and divers disease, is a form of gas embolism.
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