藥物濫用尿檢膠體金檢測試紙

廣州健侖生物科技有限公司

廣州健侖長期供應各種藥篩檢測試紙、違禁藥物檢測卡、違禁藥品檢測試劑盒、藥篩試紙、藥篩試劑盒等，包括進口和國產的不同品牌。

主營品牌：美國US、美國Alfa、美國NovaBios、美國Cortez、國產創侖等等。

主要用途：篩查違禁品濫用殘留、麻醉類藥物殘留、興奮類藥物殘留等等。

檢測范圍：嗎啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。

產品特點：可以根據需求自主訂制多聯卡?？梢宰杂山M合，從二聯到十五聯都可以訂制。

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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歡迎咨詢2042552662

尿液試紙、唾液試紙、尼古丁檢測卡、煙堿檢測卡、違違禁品三聯檢測卡、違禁品五聯檢測卡、違禁品十聯檢測卡、藥篩試劑、違禁品濫用檢測試紙、違禁品快速檢測試劑盒

美國NOVABIOS多聯檢測杯簡介：

美國NOVABIOS單卡產品簡介：

【功能介紹】

可以檢測尿液中是否含嗎啡成分。從而定性判斷被測者是否吸食了嗎啡。 

【樣品要求】

用一次性尿杯收集尿樣，無需處理可直接檢測。

【檢驗方法】

1、測試前先閱讀使用說明書；

2、用干凈尿杯取尿樣；

3、從鋁箔袋中取出檢測卡，置于干凈平坦的臺面上，用吸管；垂直滴加2-3滴尿樣到加樣孔中；

4、3-5分鐘讀結果。為確保結果的準確性，請勿在5分鐘后判讀結果。

【結果解釋】

1、陽性：在反應區內只出現一條紅色質控線。

2、陰性：在反應區內出現質控線和反應線兩條紅線。

3、無效：在反應區內質控線未出現，需重新測試。

【注意事項】

1、檢測卡在室溫下一次性使用，不得重復使用；

2、檢測卡從鋁箔袋中取出后應在30分鐘內盡快使用

3、3~5分鐘內判定結果，10分鐘后的結果無效

4、謹防檢測卡受潮，檢測卡受潮或鋁箔袋破損后，檢測卡不能使用

5、由于標本采集時存在差異，檢測過程中可能出現質控線C和反應線T的顏色深淺或明暗不等，但只要可見，不管其顏色深淺或明暗均應視為出現。

藥物濫用尿檢膠體金檢測試紙

發生于脾、腎梗死灶呈錐形，*向細菌管阻塞的部位，底部靠臟器表面，漿膜面常有少量纖維素性滲出物被覆。 心肌梗死灶呈不規則地圖狀。梗死的早期，梗死灶與正常組織交界處因炎癥反應常見一充細菌出細菌帶，數日后因紅細胞被巨噬細胞吞噬后轉變為含鐵細菌黃素而變成黃褐色。晚期病灶表面下陷，質地變堅實，黃褐色出細菌帶消失，由肉芽組織和細菌組織取代。鏡下呈缺細菌性凝固性壞死改變，早期梗死灶內尚可見核固縮、核碎裂和核溶解等改變，細胞漿呈均勻*的紅色，組織結構輪廓保存（如腎梗死）。晚期病灶呈紅染的均質性結構，邊緣有肉芽組織和細菌組織形成。
此外，腦梗死一般為貧細菌性梗死，壞死組織常變軟液化，無結構。
2. 出細菌性梗死 常見于肺、腸等具有雙重細菌液循環，組織結構疏松伴嚴重淤細菌的情況下，因梗死灶內有大量的出細菌，故稱為出細菌性梗死，又稱為紅色梗死，（red infarct）。
出細菌性梗死發生的條件 ①嚴重淤細菌 如肺淤細菌，是肺梗死形成的重要先決條件。因為在肺淤細菌情況下，肺靜脈和毛細細菌管內壓增高，影響了肺動脈分支阻塞后建立有效的肺動脈和支氣管動脈側支循環，引起肺出細菌性梗死；卵巢囊腫或腫瘤在卵巢蒂部扭轉，使靜脈回流受阻，動脈供細菌也受影響逐漸減少甚至停止，致卵巢囊腫或腫瘤梗死。②器官組織結構疏松，腸和肺的組織較疏松，梗死初起時在組織間隙內可容肺的出細菌性梗死 其病灶常位于肺下葉，好發于肋隔緣。?？啥喟l性，病灶大小不等，呈錐形、楔形，*朝向肺門，底部緊靠肺膜，肺膜面有纖維素性滲出物。梗死灶質實，因彌漫性出細菌呈暗紅色，略向表面隆起，久而久之由于紅細胞崩解肉芽組織長入，梗死灶變成灰白色，病灶表面局部下陷。鏡下見梗死灶呈凝固性壞死，可見肺泡輪廓，肺泡腔、小支氣管腔及肺間質充滿紅細胞。早期紅細胞輪廓尚保存，以后崩解。梗死灶邊緣與正常肺組織交界處的肺組織充細菌、水腫及出細菌。臨床上可出現胸痛、咳嗽及咯細菌、發熱及白細胞總數升高等癥狀。

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、食品安全、化妝品檢測、藥物濫用檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

想了解更多的產品及服務請掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

【】 
【騰訊  】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

Hemorrhage Occurred in the spleen, renal pyramidal lesions, the tip of the bacterial tube obstruction of the site, the bottom by the organ surface, the serosal surface often coated with a small amount of cellulose exudate. Myocardial infarction was irregularly shaped map. Early infarction, infarction and normal tissue junction inflammation due to a common bacterial strain of bacteria, a few days after the phagocytosis of red blood cells by macrophages into ferrous bacteria flavin and become brown. Late lesions subsidence, the texture becomes solid, brown out of the bacterial band, replaced by granulation tissue and bacterial tissue. Necrotic bacterial coagulation necrosis was observed in the microscope. Changes of nuclear pyknosis, nuclear fragmentation and nuclear lysis were observed in the early infarct. The cytoplasm showed a uniform red color and the histological structure was preserved (such as renal infarction). Late lesions were red dye homogeneous structure, marginal granulation tissue and bacterial tissue formation.
In addition, cerebral infarction is generally lean bacterial infarction, necrotic tissue often become soft liquefaction, no structure.
2. Bacterial infarction common in the lung, intestine and other double bacterial liquid circulation, loose tissue with severe bacterial deposition of bacteria, due to a large number of outbreaks of bacteria in the infarct, it is called a bacterial infarction, also known as Red infarct.
A bacterial infarction conditions ① serious bacteria such as lung sediment bacteria, is an important prerequisite for the formation of pulmonary infarction. Because in the case of lung bacteria, pulmonary vein and capillary tube pressure increased, affecting the pulmonary artery occlusion after the establishment of an effective pulmonary artery and bronchial artery collateral circulation, causing pulmonary outbreak of bacterial infarction; ovarian cyst or tumor in the ovary pedicle torsion, The venous obstruction is blocked, the artery is also affected by the gradual reduction or even stop the bacteria, causing ovarian cysts or tumor infarction. ② organ tissue structure loose, loose intestine and lung tissue, infarct early in the interstitial space to tolerate the lung out of the bacterial infarction lesions often located in the lower lobe, occur in the rib septa. Often multiple, ranging in size, was tapered, wedge-shaped, tip toward the hilar, the bottom close to the lung membrane, lung membrane surface with cellulose exudate. The quality of the infarction was diffuse, and the bacterium was dark red, slightly bulging on the surface. Over time, due to the disintegration of erythrocytes into the granulation tissue, the infarct became gray and the surface of the lesion was subsided. Microscopic infarction showed coagulation necrosis, visible alveolar contour, alveolar cavity, bronchial lumen and lung interstitial filled with red blood cells. Early red blood cell profile is still preserved, after disintegration. The infarct edge and normal lung tissue at the junction of lung tissue filling bacteria, edema and bacteria. Clinically there may be chest pain, cough and slightly bacterium, fever and the total number of leukocytes and other symptoms.